Contribution to the study of Parinaud syndrome

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We reviewed the history of Parinaud syndrome, since its description in 1883. There is evidence that she has experienced a definite developments due to the current understanding of the mechanisms associated with large vertical gaze area. Also discussed are the various clinical aspects of the problem,...

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Detalles Bibliográficos
Autores: Voto Bernales, Jorge, Mejía B., Carlos A.
Formato: artículo
Fecha de Publicación:1957
Institución:Universidad Nacional Mayor de San Marcos
Repositorio:Revistas - Universidad Nacional Mayor de San Marcos
Lenguaje:español
OAI Identifier:oai:ojs.csi.unmsm:article/10719
Enlace del recurso:https://revistasinvestigacion.unmsm.edu.pe/index.php/anales/article/view/10719
Nivel de acceso:acceso abierto
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spelling Contribution to the study of Parinaud syndromeContribución al estudio del síndrome de ParinaudVoto Bernales, JorgeMejía B., Carlos A.We reviewed the history of Parinaud syndrome, since its description in 1883. There is evidence that she has experienced a definite developments due to the current understanding of the mechanisms associated with large vertical gaze area. Also discussed are the various clinical aspects of the problem, through the copious literature published after communication Parinaud. We provide 4 personal observations of Parinaud syndrome: 3 relacionodas to something more complicated neurological syndromes, and 1 similar to the classic description of Parinaud. These observations confirm the general impression of the outstanding exclusivity of this syndrome appears frequently included in more complex neurological cases. The etiology has also been different in our 4 observations; this represents the fragility of neural structures involved in vertical gaze at the various agents that damage the nervous system. The clinical course was satisfactory in 3 cases, with a significant recovery of the oculomotor disorder 2 there was 1 case fatal, and anatomical examination showed the lesions in the subthalamus-mesoencenfálica region below the posterior white commissure, including the commissural nuclei and interstitial unilaterally.Hemos revisado la historia del síndrome de Parinaud, desde su descripción en 1883. Hay evidencia de que ella ha experimentado una evolución definida debida a los actuales conocimientos acerca de una gran área relacionada con los mecanismos de la mirada vertical. También han sido discutidos los diferentes aspectos clínicos del problema, a través de la copiosa bibliografía publicada después de la comunicación de Parinaud. Aportamos 4 observaciones personales del síndrome de Parinaud: 3 relacionodas a síndromes neurológicos algo más complicados, y 1 similar a la clásica descripción de Parinaud. Estas observaciones confirman la impresión general acerca de la excepcional exclusividad de este síndrome que con mucha frecuencia aparece incluido en casos neurológicos más complicados. La etiología también ha sido diferente en nuestras 4 observaciones; ello representa la fragilidad de las estructuras nerviosas relacionadas con la mirada vertical a los variados agentes que dañan el sistema nervioso. El curso clínico fue satisfactorio en 3 casos, con una notable recuperación del trastorno óculomotor en 2 hubo 1 caso fatal, y el examen anatómico mostró las lesiones localizadas en la región subtálamo-mesoencenfálica, por debajo de la comisura blanca posterior, incluyendo los núcleos comisural e intersticial unilateralmente.Universidad Nacional Mayor de San Marcos, Facultad de Medicina Humana1957-03-18info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfhttps://revistasinvestigacion.unmsm.edu.pe/index.php/anales/article/view/1071910.15381/anales.v40i1.10719Anales de la Facultad de Medicina; Vol. 40 No. 1 (1957); 1-31Anales de la Facultad de Medicina; Vol. 40 Núm. 1 (1957); 1-311609-94191025-5583reponame:Revistas - Universidad Nacional Mayor de San Marcosinstname:Universidad Nacional Mayor de San Marcosinstacron:UNMSMspahttps://revistasinvestigacion.unmsm.edu.pe/index.php/anales/article/view/10719/9530Derechos de autor 1957 Jorge Voto Bernales, Carlos A. Mejía B.https://creativecommons.org/licenses/by-nc-sa/4.0info:eu-repo/semantics/openAccessoai:ojs.csi.unmsm:article/107192020-04-08T10:58:09Z
dc.title.none.fl_str_mv Contribution to the study of Parinaud syndrome
Contribución al estudio del síndrome de Parinaud
title Contribution to the study of Parinaud syndrome
spellingShingle Contribution to the study of Parinaud syndrome
Voto Bernales, Jorge
title_short Contribution to the study of Parinaud syndrome
title_full Contribution to the study of Parinaud syndrome
title_fullStr Contribution to the study of Parinaud syndrome
title_full_unstemmed Contribution to the study of Parinaud syndrome
title_sort Contribution to the study of Parinaud syndrome
dc.creator.none.fl_str_mv Voto Bernales, Jorge
Mejía B., Carlos A.
author Voto Bernales, Jorge
author_facet Voto Bernales, Jorge
Mejía B., Carlos A.
author_role author
author2 Mejía B., Carlos A.
author2_role author
description We reviewed the history of Parinaud syndrome, since its description in 1883. There is evidence that she has experienced a definite developments due to the current understanding of the mechanisms associated with large vertical gaze area. Also discussed are the various clinical aspects of the problem, through the copious literature published after communication Parinaud. We provide 4 personal observations of Parinaud syndrome: 3 relacionodas to something more complicated neurological syndromes, and 1 similar to the classic description of Parinaud. These observations confirm the general impression of the outstanding exclusivity of this syndrome appears frequently included in more complex neurological cases. The etiology has also been different in our 4 observations; this represents the fragility of neural structures involved in vertical gaze at the various agents that damage the nervous system. The clinical course was satisfactory in 3 cases, with a significant recovery of the oculomotor disorder 2 there was 1 case fatal, and anatomical examination showed the lesions in the subthalamus-mesoencenfálica region below the posterior white commissure, including the commissural nuclei and interstitial unilaterally.
publishDate 1957
dc.date.none.fl_str_mv 1957-03-18
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://revistasinvestigacion.unmsm.edu.pe/index.php/anales/article/view/10719
10.15381/anales.v40i1.10719
url https://revistasinvestigacion.unmsm.edu.pe/index.php/anales/article/view/10719
identifier_str_mv 10.15381/anales.v40i1.10719
dc.language.none.fl_str_mv spa
language spa
dc.relation.none.fl_str_mv https://revistasinvestigacion.unmsm.edu.pe/index.php/anales/article/view/10719/9530
dc.rights.none.fl_str_mv Derechos de autor 1957 Jorge Voto Bernales, Carlos A. Mejía B.
https://creativecommons.org/licenses/by-nc-sa/4.0
info:eu-repo/semantics/openAccess
rights_invalid_str_mv Derechos de autor 1957 Jorge Voto Bernales, Carlos A. Mejía B.
https://creativecommons.org/licenses/by-nc-sa/4.0
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.publisher.none.fl_str_mv Universidad Nacional Mayor de San Marcos, Facultad de Medicina Humana
publisher.none.fl_str_mv Universidad Nacional Mayor de San Marcos, Facultad de Medicina Humana
dc.source.none.fl_str_mv Anales de la Facultad de Medicina; Vol. 40 No. 1 (1957); 1-31
Anales de la Facultad de Medicina; Vol. 40 Núm. 1 (1957); 1-31
1609-9419
1025-5583
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