We simulated the dynamics of CO2 transport in the alveolar sacs of the human lung. Using Arbitrary Lagrangian Eulerian (ALE) framework, we control the movement domain for a normal and fast maneuver. The fluid of room air inspired and CO2 concentrations were approximated by Navier-Stokes and convection diffusion equations; the stress-stretch in the wall for different volumes were quantified in equal time of respiration. The expansion for a normal and forced maneuver were represented as 9 and 90% to the initial geometry. The difference of the CO2 was 73x10

We simulated the dynamics of CO2 transport in the alveolar sacs of the human lung. Using Arbitrary Lagrangian Eulerian (ALE) framework, we control the movement domain for a normal and fast maneuver. The fluid of room air inspired and CO2 concentrations were approximated by Navier-Stokes and convection diffusion equations; the stress-stretch in the wall for different volumes were quantified in equal time of respiration. The expansion for a normal and forced maneuver were represented as 9 and 90% to the initial geometry. The difference of the CO2 was 73x10

In this article, we formulate a physiological process through a two-dimensional fluid-structure interaction problem between the airflow and the pulmonary alveolus in the Eulerian-Lagrangian Arbitrary (ALE) frame.This problem arises by coupling the equations of the fluid and the structure, described by the Navier-Stokes equations of evolution for incompressible flows, and an equilibrium equation, respectively.

In this article, we formulate a physiological process through a two-dimensional fluid-structure interaction problem between the airflow and the pulmonary alveolus in the Eulerian-Lagrangian Arbitrary (ALE) frame.This problem arises by coupling the equations of the fluid and the structure, described by the Navier-Stokes equations of evolution for incompressible flows, and an equilibrium equation, respectively.